Related Articles

Jekaterina Erenpreisa, Kristine Salmina, Anda Huna, Thomas R. Jackson, Alejandro Vazquez-Martin and Mark S. Cragg. The “virgin birth”, polyploidy, and the origin of cancer. Oncoscience 2015; 2 (1): 3-14.

Link: http://www.impactjournals.com/oncoscience/files/papers/1/108/108.pdf

 

David Dı´az-Carballo,  Sebastian Gustmann,  Holger Jastrow,  Ali Haydar Acikelli,  Philip Dammann, Jacqueline Klein,  Ulrike Dembinski,  Walter Bardenheuer,  Sascha Malak,  Marcos J. Arau´ zo-Bravo, Beate Schultheis,  Constanze Aldinger,  and Dirk StrumbergAtypical. Cell Populations Associated with Acquired Resistance to Cytostatics and Cancer Stem Cell Features: AU1 c The Role of Mitochondria in Nuclear Encapsulation. 2014; DNA AND CELL BIOLOGY, 33 (11): 1–26.

Link: http://online.liebertpub.com/doi/abs/10.1089/dna.2014.2375

Erenpreisa J, Cragg M. MOS, aneuploidy and the ploidy cycle of cancer cells. Oncogene 2010; 29(40): 5447–51. Link: http://www.ncbi.nlm.nih.gov/pubmed/20676137

 

Erenpreisa J, Cragg M. Three steps to the immortality of cancer cells: senescence, polyploidy and self-renewal. Cancer Cell Int. 2013; 13(1): 92 Link: http://www.cancerci.com/content/13/1/92

 

Tanabe BK, Takahashi K, Yamanaka S. Induction of pluripotency by defined factors. Proc. Jpn. Acad., 2014; Ser. B 90; 83-96. Link: http://www.ncbi.nlm.nih.gov/pmc/articles/PMC3997808/

 

Zhang S, Mercado-Uribe I, Liu J. Tumor stroma and differentiated cancer cells can be originated directly from polyploid giant cancer cells induced by paclitaxel. Int J Cancer. 2014; 134(3):508-18. Link: http://www.pubfacts.com/detail/23754740/Tumor-stroma-and-differentiated-cancer-cells-can-be-originated-directly-from-polyploid-giant-cancer-

 

Buganim Y, Faddah DA, Jaenisch R. Mechanisms and models of somatic cell reprogramming. Nature Reviews Genetics 2013; 14: 427-439. Link: http://connection.ebscohost.com/c/articles/87656259/mechanisms-models-somatic-cell-reprogramming

 

Forsberg LA, Rasi C, Malmqvist N, Davies H, Pasupulati S, Pakalapati G, Sangren J, Diaz de Stahl T, Zaghlool A, at al. Mosaic loss of chromosome Y in peripheral blood is associated with shorter survival and higher risk of cancer. Nature Genetics 2014; 46: 624-628. Link: http://www.nature.com/ng/journal/v46/n6/abs/ng.2966.html

 

Ya-ling Tang, Yun-long Fan, Jian Jiang, Kai-de Li, Min Zheng, Wei Chen,
Xiang-rui Ma, Ning Geng, Qian-ming Chen, Yu Chen, Xin-hua Liang. C-kit induces epithelial–mesenchymal transition and contributes to salivary adenoid cystic cancer progression. Oncotarget, 2014; Vol. 5, No. 6, pp. 1491 – 1501. Link: http://www.ncbi.nlm.nih.gov/pmc/articles/PMC4039226/

 

Wang LCao JJi PZhang DMa LDym MYu ZFeng L. Oocyte-like cells induced from mouse spermatogonial stem cells. Cell Biosci. 2012 Aug 6; 2(1):27. doi: 10.1186/2045-3701-2-27.http://www.cellandbioscience.com/content/2/1/27

 

Aurora M. Nedelcu, Oana Marcu and Richard E. Michod. Sex as a response to oxidative stress: a twofold increase in cellular reactive oxygen species activates sex genes. Proc. R. Soc. Lond. B 2004 271, 1591-1596. Link: http://rspb.royalsocietypublishing.org/content/271/1548/1591#related-urls

 

Adams IR,  McLaren A. Sexually dimorphic development of mouse primordial germ cells: switching from oogenesis to spermatogenesis. Development. 2002 Mar;129(5):1155-64. Link: http://dev.biologists.org/content/129/5/1155.long

 

Anfei Liu, Xiya Yu, and Shanrong Liu. Pluripotency transcription factors and cancer stem cells: small genes make a big difference. Chin J Cancer. 2013 Sep;32(9):483-7. doi: 10.5732/cjc.012.10282. Link: http://www.ncbi.nlm.nih.gov/pubmed/23419197

 

Nagata S, Hirano K, Kanemori M, Sun L-T, Tada T (2012) Self-Renewal and Pluripotency Acquired through Somatic Reprogramming to Human Cancer Stem Cells. PLoS ONE 7(11): e48699. doi:10.1371/journal.pone.0048699. Link: http://www.plosone.org/article/info%3Adoi%2F10.1371%2Fjournal.pone.004869

 

Bussard KM, Smith GH. Human Breast Cancer Cells Are Redirected to Mammary Epithelial Cells upon Interaction with the Regenerating Mammary Gland Microenvironment In-Vivo. PLoS ONE 2012; 7(11): e49221. doi: 10.1371/journal.pone.0049221 Link: http://www.plosone.org/article/info%3Adoi%2F10.1371%2Fjournal.pone.0049221

 

Walen KH. Normal Human Cells Acquiring Proliferative Advantage to Hyperplasia-Like Growth-Morphology: Aberrant Progeny Cells Associated with Endopolyploid and Haploid Divisions. Cancer and Clinical Oncology 2013; Vol. 2, No. 2, 1-15. Link: http://www.ccsenet.org/journal/index.php/cco/article/view/28704

 

Walen KH. Neoplastic-Like CELL Changes of Normal Fibroblast Cells Associated with Evolutionary Conserved Maternal and Paternal Genomic Autonomous Behavior (Gonomery). 2014; Journal of Cancer Therapy, 2014, 5, 860-877. http://dx.doi.org/10.4236/jct.2014.59094

Link: http://www.scirp.org/journal/PaperInformation.aspx?paperID=49182#.VFaS6zTF98E

 

Walen KH. Haploidization of Human Diploid Metaphase Cells: Is This Genome Reductive Mechanism Opperational in Near-Haploid Leukemia? 2014; Journal of Cancer Therapy, 2014, 5, 101-114

Link: http://www.scirp.org/journal/PaperInformation.aspx?paperID=42123#.VFaTlDTF98E